Feds want five-year paper trail for livestock
By Megan R. Wilson - 05/06/13 01:59 PM ET
The Obama administration has adopted stricter record keeping rules for the
livestock industry in hopes of aiding officials who trace animal-borne diseases.
First proposed by the Animal and Plant Health Inspection Service (APHIS)
under the George W. Bush administration, the regulations require livestock
producers, slaughterhouses and rendering plants to keep five years of records
for animals that have been in their facilities.
The previous rules only required two years of recordkeeping and excluded
the slaughtering and rendering industries.
The inspection service wants access to documents such as “weight tickets,
sales slips, and records of origin, identification, and destination that relate
to livestock that are in, or that have been in, the facility,” according to a
Federal Register publication scheduled to print on Tuesday.
The proposal, released in 2008, received little feedback from the public or
the industry.
One comment complained that slaughterhouses and rendering plants are
already subject to record keeping requirements under the Food and Drug
Administration, but the inspection service countered that those records only
need to be kept for one year.
Some diseases, such as bovine tuberculosis, can stay dormant for months or
years, the agency says.
It wants to be able to go through records and track the source of an
illness long after an animal dies.
Since poultry and pigs have a shorter lifespan than other livestock, APHIS
agreed to only require a two-year retention of documents for those animals.
The regulations will become effective 30 days after publication in the
Federal Register.
it’s about damn time !
I wonder if this includes cervids as well ? according to the shooting pen
owners and USDA et al, cervids is their livestock now $$$
Wednesday, April 24, 2013
Dissociation between Transmissible Spongiform Encephalopathy (TSE)
Infectivity and Proteinase K-Resistant PrPSc Levels in Peripheral Tissue from a
Murine Transgenic Model of TSE Disease
Saturday, December 15, 2012
Bovine spongiform encephalopathy: the effect of oral exposure dose on
attack rate and incubation period in cattle -- an update 5 December 2012
Friday, April 19, 2013
FDA BSE TSE PRION NEWS FEED AND ANNUAL INSPECTION OF FEED MILLS REPORTS HAS
CEASED TO EXIST
Monday, March 25, 2013
Minnesota Firm Recalls Bone-In Ribeye That May Contain Specified Risk
Materials Recall Release CLASS II RECALL FSIS-RC-024-2013
Tuesday, March 5, 2013
Use of Materials Derived From Cattle in Human Food and Cosmetics; Reopening
of the Comment Period FDA-2004-N-0188-0051 (TSS SUBMISSION)
FDA believes current regulation protects the public from BSE but reopens
comment period due to new studies
Wednesday, March 20, 2013
GAO-13-244, Mar 18, 2013 Dietary Supplements FDA May Have Opportunities to
Expand Its Use of Reported Health Problems to Oversee Product
From: Terry S. Singeltary Sr.
Sent: Tuesday, March 19, 2013 2:46 PM
To: gomezj@gao.gov
Cc: siggerudk@gao.gov ; youngc1@gao.gov ; oighotline@gao.gov
Wednesday, February 20, 2013
World Organization for Animal Health Recommends United States' BSE Risk
Status Be Upgraded
Statement from Agriculture Secretary Tom Vilsack:
Thursday, February 14, 2013
The Many Faces of Mad Cow Disease Bovine Spongiform Encephalopathy BSE and
TSE prion disease
Friday, April 19, 2013
Bovine Spongiform Encephalopathy (BSE) Feed Safety Support Program Grants
Fiscal Year 2011: October 1, 2010 - September 30, 2011 FDA
Friday, April 19, 2013
APHIS 2013 Stakeholder Meeting (March 2013) BSE TSE PRION
Tuesday, April 30, 2013
Mad cow infected blood 'to kill 1,000’
Wednesday, April 24, 2013
Chimpanzees Released After 30 Years Of Testing, Brace Yourself For Smiles
Tuesday, April 30, 2013
Foodborne Transmission of Bovine Spongiform Encephalopathy to Nonhuman
Primates
Monday, May 6, 2013
BSE-associated Prion-Amyloid Cardiomyopathy in Primates
Volume 19, Number 6—June 2013
Tuesday, December 15, 2009
NAIS, COOL, FROM FARM TO FORK, MAD COW DISEASE
Greetings,
I do not understand the logic behind the fear to NAIS and COOL ?
I really don't.
Everything about us is traceable by the Government. Birth certificate,
driver's license, social security number, your deed to your house, property,
house number, car title, all the parts of your car are traceable, even each
owner of the car since it rolled off the assembly line, boat, motor, etc.
etc.
SO why all the concern on traceability of the food we eat ?
WHY does most folks in the industry fear this so much $$$
The fear of the NAIS and or COOL seems to be that no one wants their
product to be traceable, because of fear of litigation from contaminated food,
rather than the excuse of not wanting the Government messing in your business.
Just my opinion.
An example would be the California mad cow beef recall (see history below),
that eventually humans were exposed to suspect mad cow beef. A lot of folks.
Same as with the Dead Stock Downer Cow School Lunch Program. These kids all
across our Nation, for four years and probably longer, were fed the most high
risk cattle for BSE aka mad cow disease from dead stock downer cows, where the
largest beef recall (at the time) took place, of which, a great deal were
already consumed by our children. With an incubation period of 50+ years in some
cases, in others much shorter, who will watch our children for CJD for the next
5+ decades ?
snip...see full text ;
Friday, August 20, 2010 USDA: Animal Disease Traceability August 2010 USDA:
Animal Disease Traceability August 2010
Greetings,
as a consumer, my opinion (if that matters), you need some sort of
traceability for your livestock. I don't want to know anything about your
family, your income, your kids, your sex life, nothing but to be able to trace
that animal from farm to fork. I want to know whether or not if that animal has
been fed animal protein, antibiotics, and if myself or my family do get sick, we
should be able to trace that product. I don't see the problem. you can trace
every part on your car, I can find out anything I want about you on the
internet. why can't I do that with a cow?
let's review a few things about the NAIS, why we need it past and present.
let's review first the few mad cows that were accidently found, birth cohorts,
herd cohorts etc., and how efficient or NOT the traceability of those 4 animals
were....oops, that's right, the first stumbling and staggering mad cow in Texas
was sent straight to the render, did not pass GO(inspection), did not get
$200.(prion rapid test), and of course, was never confirmed. THEN the second mad
cow, the one that would never have been confirmed if not for an act of Congress
and the Honorable Fong of the OIG, and scientist from the EU and the USA asking
questions, meanwhile the BSE MRR was being finalized. Then after 7 months of
sitting on the shelf, after a secret test had already turned up positive, but
yet still 7 months no confirmation on a BSE positive test that by the BSE Red
Books, it should have been a 48 hour turnaround on that test. Finally, the 2nd
Texas mad cow was confirmed positive BSE. later termed atypical h-BSE. Then you
had the Alabama mad cow, now termed g-h-BSEalabama, and then of course the mad
cow old Luther capped in Washington state, the white one, that changed colors,
and then was said to be a Canadian traitor c-BSE mad cow. IT's all Canada's
fault ;-(NOT)
Be sure to see the latest data on the typical and atypical cases of BSE and
Scrapie and any human CJD TSE there from. This is down toward the bottom of the
posting.
BSE, USDA, NAIS, AND TRACEABILITY
Let's look at how the USDA et al trace BSE aka mad cow cases, birth, and
index herd cattle in the past (or rather how they could not trace them).
TEXAS MAD COW (h-BSE), that was finally tested and documented 7+ months
after an act of Congress, and Scientist from all over the Globe questioning the
testing methods and negative findings of this Texas mad cow. ...
TEXAS h-BSE MAD COW CASE THAT WAS FINALLY DOCUMENTED
snip...see full text ;
February 10, 2010
Greetings,
IN my opinion, i think we need NAIS. i think as a consumer, we have a right
to this information. as a Country importing our product, they have a right to
know, it should be law. it should be mandatory that when an animal disease or
human disease there from break out, the animal and or it's product can be
traced. and as a producer of that product, if you are too worried about
confidentiality, you are trying to hide something, then you should not be in the
business. how in the world can knowing from where a cow comes from i.e.
traceability, be such a threat to the producer, unless they simply want to hide
something? in 2010, the USA, in relations to cattle identification and tracing,
could not trace their @ss if they had both hands on it, in my opinion, and going
by past history of the last two documented mad cows. the USA has been discussing
this for over a decade. I don't understand it, all these other Country's that
have some sort of Animal Traceability in place, they are and have been trying to
eradicate BSE, that have had a feed ban in place, that have been abiding by it,
testing in numbers to find and eradicate the TSEs, and the USA just seems to be
doing the opposite in many ways it seems to me. if the USA is not going to trace
it's meat products, why should other Country's trace theirs? The USA is BSE GBR
III, all of North America is BSE GBR III (with all the evidence of breaches in
the bse feed ban, the breaches in the BSE surveillance program, i personally
believe it is BSE GBR IV). The problem is, the USDA just never accepted it (BSE
GBR III), and then changed the rules with the BSE MRR. this BSE MRR policy
literally trashed 30 years of attempted BSE GBR eradication of this disease.
What about other trading partners with the U.S.A. that DO HAVE a traceability
system. i think Australia is getting ready to roll over and get MRR'ed,
therefore they too will just be another victim of allowing all strains of
Typical and Atypical BSE/TSE into their Country via the USDA OIE BSE MRR policy,
a policy of trading all strains of mad cow disease globally. The O.I.E., by
bending over for the USDA with this damn BSE MRR policy, has sold their sole to
the devil, and in doing so, sold yours too. ...
remember, and do not forget what the Honorable Nobel Peace Prize winner of
the PRION i.e. Stanley Prusiner said ;
THEY DON'T WANT TO KNOW ! ABSOLUTE IGNORANCE, ALL THAT MATTERS IS
TRADE...
AFTER THE COW IN CANADA...LEVEL OF ABSOLUTE IGNORANCE, OF WHAT HE WAS
TRYTING TO CONTAIN...
THE ENTIRE POLICY WAS DRIVEN BY WHAT THE USA WAS TELLING HIM TO DO...SO NOW
AFTER SOME TIME HAS PASSED...
SO NOW AFTER TIME HAS PASSED IT'S O.K. FOR BONELESS BEEF PRODUCTS FROM
UNDER 30 MONTHS TO BE EXPORTED FROM CANADA TO THE UNITED STATES, THAT'S ALL THAT
MATTERED...
YES, I THINK THAT PRIONS ARE BAD TO EAT, AND YOU CAN DIE FROM THEM...
snip...see full text ;
Friday, December 21, 2012
USDA Issues Final Rule for Animal Disease Traceability
Friday, March 13, 2009
NAIS comments NCBA and R-Calf Wednesday, March 11, 2009 – 10:30 a.m.
Subcommittee on Livestock, Dairy, and Poultry — Public Hearing
Saturday, August 16, 2008
Qualitative Analysis of BSE Risk Factors in the United States February 13,
2000 at 3:37 pm PST (BSE red book)
48 hour traceback for BSE mad cow disease in the USA ???
NOT in your lifetime !
8 YEARS IN REVIEW OF THE MAD COW DEBACLE IN THE USA ;
Tuesday, April 16, 2013
Cervid Industry Unites To Set Direction for CWD Reform and seem to ignore
their ignorance and denial in their role in spreading Chronic Wasting
Disease
Thursday, May 02, 2013
Chronic Wasting Disease (CWD) Texas Important Update on OBEX ONLY TEXTING
Thursday, February 21, 2013
National Prion Disease Pathology Surveillance Center Cases Examined January
16, 2013
16 YEAR OLD SPORADIC FFI ?
Monday, January 14, 2013
Gambetti et al USA Prion Unit change another highly suspect USA mad cow
victim to another fake name i.e. sporadic FFI at age 16 CJD Foundation goes
along with this BSe
Monday, December 31, 2012
Creutzfeldt Jakob Disease and Human TSE Prion Disease in Washington State,
2006–2011-2012
Tuesday, December 25, 2012
CREUTZFELDT JAKOB TSE PRION DISEASE HUMANS END OF YEAR REVIEW DECEMBER 25,
2012
Tuesday, June 26, 2012
Creutzfeldt Jakob Disease Human TSE report update North America, Canada,
Mexico, and USDA PRION UNIT as of May 18, 2012
type determination pending Creutzfeldt Jakob Disease (tdpCJD), is on the
rise in Canada and the USA
Wednesday, June 13, 2012
MEXICO IS UNDER or MIS DIAGNOSING CREUTZFELDT JAKOB DISEASE AND OTHER PRION
DISEASE SOME WITH POSSIBLE nvCJD
*** The discovery of previously unrecognized prion diseases in both humans
and animals (i.e., Nor98 in small ruminants) demonstrates that the range of
prion diseases might be wider than expected and raises crucial questions about
the epidemiology and strain properties of these new forms. We are investigating
this latter issue by molecular and biological comparison of VPSPr, GSS and
Nor98.
VARIABLY PROTEASE-SENSITVE PRIONOPATHY IS TRANSMISSIBLE ...price of prion
poker goes up again $
OR-10: Variably protease-sensitive prionopathy is transmissible in bank
voles
Romolo Nonno,1 Michele Di Bari,1 Laura Pirisinu,1 Claudia D’Agostino,1
Stefano Marcon,1 Geraldina Riccardi,1 Gabriele Vaccari,1 Piero Parchi,2 Wenquan
Zou,3 Pierluigi Gambetti,3 Umberto Agrimi1 1Istituto Superiore di Sanità; Rome,
Italy; 2Dipartimento di Scienze Neurologiche, Università di Bologna; Bologna,
Italy; 3Case Western Reserve University; Cleveland, OH USA
Background. Variably protease-sensitive prionopathy (VPSPr) is a recently
described “sporadic”neurodegenerative disease involving prion protein
aggregation, which has clinical similarities with non-Alzheimer dementias, such
as fronto-temporal dementia. Currently, 30 cases of VPSPr have been reported in
Europe and USA, of which 19 cases were homozygous for valine at codon 129 of the
prion protein (VV), 8 were MV and 3 were MM. A distinctive feature of VPSPr is
the electrophoretic pattern of PrPSc after digestion with proteinase K (PK).
After PK-treatment, PrP from VPSPr forms a ladder-like electrophoretic pattern
similar to that described in GSS cases. The clinical and pathological features
of VPSPr raised the question of the correct classification of VPSPr among prion
diseases or other forms of neurodegenerative disorders. Here we report
preliminary data on the transmissibility and pathological features of VPSPr
cases in bank voles.
Materials and Methods. Seven VPSPr cases were inoculated in two genetic
lines of bank voles, carrying either methionine or isoleucine at codon 109 of
the prion protein (named BvM109 and BvI109, respectively). Among the VPSPr cases
selected, 2 were VV at PrP codon 129, 3 were MV and 2 were MM. Clinical
diagnosis in voles was confirmed by brain pathological assessment and western
blot for PK-resistant PrPSc (PrPres) with mAbs SAF32, SAF84, 12B2 and 9A2.
Results. To date, 2 VPSPr cases (1 MV and 1 MM) gave positive transmission
in BvM109. Overall, 3 voles were positive with survival time between 290 and 588
d post inoculation (d.p.i.). All positive voles accumulated PrPres in the form
of the typical PrP27–30, which was indistinguishable to that previously observed
in BvM109 inoculated with sCJDMM1 cases.
In BvI109, 3 VPSPr cases (2 VV and 1 MM) showed positive transmission until
now. Overall, 5 voles were positive with survival time between 281 and 596
d.p.i.. In contrast to what observed in BvM109, all BvI109 showed a GSS-like
PrPSc electrophoretic pattern, characterized by low molecular weight PrPres.
These PrPres fragments were positive with mAb 9A2 and 12B2, while being negative
with SAF32 and SAF84, suggesting that they are cleaved at both the C-terminus
and the N-terminus. Second passages are in progress from these first successful
transmissions.
Conclusions. Preliminary results from transmission studies in bank voles
strongly support the notion that VPSPr is a transmissible prion disease.
Interestingly, VPSPr undergoes divergent evolution in the two genetic lines of
voles, with sCJD-like features in BvM109 and GSS-like properties in BvI109.
The discovery of previously unrecognized prion diseases in both humans and
animals (i.e., Nor98 in small ruminants) demonstrates that the range of prion
diseases might be wider than expected and raises crucial questions about the
epidemiology and strain properties of these new forms. We are investigating this
latter issue by molecular and biological comparison of VPSPr, GSS and Nor98.
Wednesday, March 28, 2012
VARIABLY PROTEASE-SENSITVE PRIONOPATHY IS TRANSMISSIBLE, price of prion
poker goes up again $
Sunday, March 31, 2013
Creutzfeldt Jakob Disease CJD worlds youngest documented victim, 11 years
old, shall we pray
Monday, April 15, 2013
Dr. Stephen B. Thacker Director Centers for Disease Control and
Prevention′s Office of Science, Epidemiology and Laboratory Services (OSELS)
dies from Creutzfeldt Jakob Disease CJD
IT is of my opinion, that the OIE and the USDA et al, are the soul reason,
and responsible parties, for Transmissible Spongiform Encephalopathy TSE prion
diseases, including typical and atypical BSE, typical and atypical Scrapie, and
all strains of CWD, and human TSE there from, spreading around the globe.
I have lost all confidence of this organization as a regulatory authority
on animal disease, and consider it nothing more than a National Trading
Brokerage for all strains of animal TSE, just to satisfy there commodity. AS i
said before, OIE should hang up there jock strap now, since it appears they will
buckle every time a country makes some political hay about trade protocol,
commodities and futures. IF they are not going to be science based, they should
do everyone a favor and dissolve there organization.
JUST because of low documented human body count with nvCJD and the long
incubation periods, the lack of sound science being replaced by political and
corporate science in relations with the fact that science has now linked some
sporadic CJD with atypical BSE and atypical scrapie, and the very real threat of
CWD being zoonosis, I believed the O.I.E. has failed terribly and again, I call
for this organization to be dissolved. ...
A_Annex_VII_A_AHG_Report_Meeting_Final
Scrapie – The disease does not show significant morbidity (2-30%
within-flock morbidity) or mortality and is not zoonotic. However, the Group
noted the difficulty in evaluating the level of morbidity for diseases with a
long incubation period such as scrapie. The Group recommended that the disease
be delisted.
Chapter 1.6.
PROCEDURES FOR SELF DECLARATION AND FOR OFFICIAL RECOGNITION BY the
OIE
Article 1.6.1.
[No change]
Article 1.6.2.
[No change]
Article 1.6.3.
Questionnaire on bovine spongiform encephalopathy
SNIP...
Article 11.5.29.
Conclusions of the risk assessment
The overall risk of BSE in the cattle population of a country or zone is
proportional to the level of known or potential exposure to BSE infectivity and
the potential for recycling and amplification of the infectivity through
livestock feeding practices. For the risk assessment to conclude that the cattle
population of a country or zone is free from BSE risk, it should have
demonstrated that appropriate measures have been taken to manage any risks
identified.
BOUGHT AND PAID FOR BY YOUR LOCAL CATTLE DEALERS AND LOBBYIST EVERYWHERE...
IN A NUT SHELL ;
(Adopted by the International Committee of the OIE on 23 May 2006)
11. Information published by the OIE is derived from appropriate
declarations made by the official Veterinary Services of Member Countries. The
OIE is not responsible for inaccurate publication of country disease status
based on inaccurate information or changes in epidemiological status or other
significant events that were not promptly reported to the Central Bureau,
Tuesday, July 17, 2012
O.I.E. BSE, CWD, SCRAPIE, TSE PRION DISEASE Final Report of the 80th
General Session, 20 - 25 May 2012
Wednesday, May 25, 2011
O.I.E. Terrestrial Animal Health Standards Commission and prion (TSE)
disease reporting 2011
----- Original Message -----
From: Terry S. Singeltary Sr.
To: BSE-L@LISTS.AEGEE.ORG
Cc: trade@oie.int ; oie@oie.int ; f.diaz@oie.int ; scientific.dept@oie.int
; cjdvoice@yahoogroups.com ; BLOODCJD@YAHOOGROUPS.COM
Sent: Tuesday, May 24, 2011 2:24 PM
Subject: O.I.E. Terrestrial Animal Health Standards Commission and prion
(TSE) disease reporting 2011
Saturday, December 18, 2010
OIE Global Conference on Wildlife Animal Health and Biodiversity -
Preparing for the Future (TSE AND PRIONS) Paris (France), 23-25 February 2011
Monday, November 23, 2009
BSE GBR RISK ASSESSMENTS UPDATE NOVEMBER 23, 2009 COMMISSION OF THE
EUROPEAN COMMUNITIES AND O.I.E. COMMISSION DECISION of 11 November 2009 amending
the Annex to Decision 2007/453/EC as regards the BSE status of Chile, Colombia
and Japan (notified under document C(2009) 8590)
Tuesday, January 1, 2008
BSE OIE USDA
Subject: OIE BSE RECOMMENDATION FOR USA, bought and paid for by your local
cattle dealers i.e. USDA
Date: May 14, 2007 at 9:00 am PST
OIE BSE RECOMMENDATION FOR USA, bought and paid for by your local cattle
dealers i.e. USDA
STATEMENT BY DR. RON DEHAVEN REGARDING OIE RISK RECOMMENDATION
March 9, 2007
Tuesday, November 02, 2010
IN CONFIDENCE
The information contained herein should not be disseminated further except
on the basis of "NEED TO KNOW".
BSE - ATYPICAL LESION DISTRIBUTION (RBSE 92-21367) statutory (obex only)
diagnostic criteria CVL 1992
Thursday, May 02, 2013
Chronic Wasting Disease (CWD) Texas Important Update on OBEX ONLY TEXTING
2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006
Comments on technical aspects of the risk assessment were then submitted to
FSIS.
Comments were received from Food and Water Watch, Food Animal Concerns
Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S.
Singeltary.
This document provides itemized replies to the public comments received on
the 2005 updated Harvard BSE risk assessment. Please bear the following points
in mind:
Owens, Julie
From: Terry S. Singeltary Sr. [flounder9@verizon.net]
Sent: Monday, July 24, 2006 1:09 PM
To: FSIS RegulationsComments
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE)
Page 1 of 98
FSIS, USDA, REPLY TO SINGELTARY
U.S.A. 50 STATE BSE MAD COW CONFERENCE CALL Jan. 9, 2001
Friday, April 19, 2013
APHIS 2013 Stakeholder Meeting (March 2013) BSE TSE PRION
National Scrapie Eradication Program for March 2013
The monthly report for the National Scrapie Eradication Program for March
2013 has been released. The monthly reports are now available in both PowerPoint
and PDF formats.
(PowerPoint version)
(PDF version)
Highlights of the March 2013 Report
At the end of FY 2012, the percent of cull sheep found positive at
slaughter and adjusted for face color was 0.006 percent. This measure of
prevalence has decreased by 96 percent since slaughter surveillance started in
FY 2003. As of March 31, 2013 this measure of prevalence is 0.006 percent.
Additionally, in FY 2012, 8 new infected or source flocks in seven states
were identified. This represented a 47 percent reduction in the number of new
infected and source flocks identified as compared to FY 2011. To date, two
source flocks and one infected flock have been designated for FY 2013 as of
March 31. snip... Does not include Nor98-like scrapie cases found through RSSS
(2 in FY 2007, 1 in FY 2008, 4 in FY 2010, 1 in FY 2011).
Tuesday, April 30, 2013
Transmission of classical scrapie via goat milk
Veterinary Record2013;172:455 doi:10.1136/vr.f2613
TSS
